Is Senescence the Missing Link in Postpartum Hair Loss?
4 min read
February 20,2025
LONGEVITY
Is Senescence the Missing Link in Postpartum Hair Loss?
4 min read
February 20,2025
LONGEVITY
Postpartum hair loss is often dismissed as “normal,” but what if there’s more to the story? For decades, women have been told that shedding hair after pregnancy is just a side effect of rapidly shifting hormone levels. However, emerging research points to the possibility of an additional pathophysiology involving stress-induced follicular aging and cellular senescence.
Postpartum hair loss is often dismissed as “normal,” but what if there’s more to the story? For decades, women have been told that shedding hair after pregnancy is just a side effect of rapidly shifting hormone levels. However, emerging research points to the possibility of an additional pathophysiology involving stress-induced follicular aging and cellular senescence.
01
Pathophysiology of Postpartum Hair Loss
Postpartum hair loss, often referred to as postpartum telogen effluvium (PPTE), is a prevalent but temporary form of hair loss that affects approximately one-third to one-half of postpartum women.[1] This condition typically emerges between two to four months after childbirth, presenting as a diffuse rapid shedding of hair that can be distressing for new mothers.[1] The prevailing medical explanation is that since estrogen keeps more follicles in the growth (anagen) phase during pregnancy, a large portion of follicles get pushed into the shedding (telogen) phase after pregnancy.[2] In most cases, hair growth normalizes within six months to one year.[3] PPTE quite closely resembles other forms of telogen effluvium triggered by major stressors like illness, surgery, or sudden weight loss, which also manifests as rapid diffuse shedding 2–4 months after the event. The resemblance indicates that postpartum hair loss could be linked to the body’s stress response to a sudden fluctuation in hormones and the physical stress of childbirth and pregnancy.[4] When added stressors like sleepless nights, potential nutrient deficiencies, and the general stress of motherhood are factored in, hair loss can continue longer than six months into what is known as chronic telogen effluvium.[3] Additionally, some women notice that hair density does not return to its pre-pregnancy fullness, suggesting additional causes for hair loss besides hormonal rebalancing.
Postpartum Hair Loss and Hair Follicle Cellular Senescence
While changes in hormones and postpartum stress explain one side of the story, emerging evidence suggests that much more is happening at the cellular level within hair follicles. While further research is needed, cellular senescence is thought to play a significant role in stress-related hair loss.[5,6] For example, CRH, a stress-regulating hormone, has been shown to drive senescence in hair follicle cells by prematurely shifting them out of the anagen (growth) phase. During pregnancy, CRH levels rise dramatically—reaching concentrations 1,000 to 10,000 times higher than in non-pregnant individuals—and are believed to contribute to the initiation of labor.[7,8]Moreover, pregnancy has been linked to a temporary acceleration of biological aging. Epigenetic studies indicate maternal biological age increases during pregnancy and partially reverses postpartum.[9] This suggests that aging pathways, including senescence, may be particularly active during pregnancy. Interestingly, women with preeclampsia, a condition that poses many serious health complications during and after pregnancy, display higher levels of systemic senescence.[10,11] Given that cellular senescence has been implicated in hair thinning and follicular dysfunction, it is plausible that pregnancy-associated senescence and CRH-induced follicle senescence may contribute to postpartum hair loss.[12,13,14]
01
Pathophysiology of Postpartum Hair Loss
Postpartum hair loss, often referred to as postpartum telogen effluvium (PPTE), is a prevalent but temporary form of hair loss that affects approximately one-third to one-half of postpartum women.[1] This condition typically emerges between two to four months after childbirth, presenting as a diffuse rapid shedding of hair that can be distressing for new mothers.[1] The prevailing medical explanation is that since estrogen keeps more follicles in the growth (anagen) phase during pregnancy, a large portion of follicles get pushed into the shedding (telogen) phase after pregnancy.[2] In most cases, hair growth normalizes within six months to one year.[3] PPTE quite closely resembles other forms of telogen effluvium triggered by major stressors like illness, surgery, or sudden weight loss, which also manifests as rapid diffuse shedding 2–4 months after the event. The resemblance indicates that postpartum hair loss could be linked to the body’s stress response to a sudden fluctuation in hormones and the physical stress of childbirth and pregnancy.[4] When added stressors like sleepless nights, potential nutrient deficiencies, and the general stress of motherhood are factored in, hair loss can continue longer than six months into what is known as chronic telogen effluvium.[3] Additionally, some women notice that hair density does not return to its pre-pregnancy fullness, suggesting additional causes for hair loss besides hormonal rebalancing.
Postpartum Hair Loss and Hair Follicle Cellular Senescence
While changes in hormones and postpartum stress explain one side of the story, emerging evidence suggests that much more is happening at the cellular level within hair follicles. While further research is needed, cellular senescence is thought to play a significant role in stress-related hair loss.[5,6] For example, CRH, a stress-regulating hormone, has been shown to drive senescence in hair follicle cells by prematurely shifting them out of the anagen (growth) phase. During pregnancy, CRH levels rise dramatically—reaching concentrations 1,000 to 10,000 times higher than in non-pregnant individuals—and are believed to contribute to the initiation of labor.[7,8]Moreover, pregnancy has been linked to a temporary acceleration of biological aging. Epigenetic studies indicate maternal biological age increases during pregnancy and partially reverses postpartum.[9] This suggests that aging pathways, including senescence, may be particularly active during pregnancy. Interestingly, women with preeclampsia, a condition that poses many serious health complications during and after pregnancy, display higher levels of systemic senescence.[10,11] Given that cellular senescence has been implicated in hair thinning and follicular dysfunction, it is plausible that pregnancy-associated senescence and CRH-induced follicle senescence may contribute to postpartum hair loss.[12,13,14]
Key Takeaways:
- Many new mothers experience postpartum hair loss, but its causes go beyond just shifting hormones. Emerging research suggests stress-related follicular aging may also play a role.
- During pregnancy, estrogen keeps more hair follicles in the growth phase, but after childbirth, the sudden drop in estrogen can push them into the shedding phase.
- When coupled with postpartum stressors like exhaustion and nutrient loss, hair shedding can persist longer, evolving into chronic telogen effluvium.
- New research suggests that stress-related cellular senescence in hair follicles may accelerate postpartum hair loss, making recovery slower for some women.
Key Takeaways:
- Many new mothers experience postpartum hair loss, but its causes go beyond just shifting hormones. Emerging research suggests stress-related follicular aging may also play a role.
- During pregnancy, estrogen keeps more hair follicles in the growth phase, but after childbirth, the sudden drop in estrogen can push them into the shedding phase.
- When coupled with postpartum stressors like exhaustion and nutrient loss, hair shedding can persist longer, evolving into chronic telogen effluvium.
- New research suggests that stress-related cellular senescence in hair follicles may accelerate postpartum hair loss, making recovery slower for some women.
References
- https://pubmed.ncbi.nlm.nih.gov/23974577/
- https://pmc.ncbi.nlm.nih.gov/articles/PMC4908443/
- https://pmc.ncbi.nlm.nih.gov/articles/PMC5676194/
- https://www.ncbi.nlm.nih.gov/books/NBK430848/
- https://pmc.ncbi.nlm.nih.gov/articles/PMC11068553/
- https://pmc.ncbi.nlm.nih.gov/articles/PMC7310274/
- https://pubmed.ncbi.nlm.nih.gov/23385670/
- https://pubmed.ncbi.nlm.nih.gov/24248185/
- https://www.cell.com/cell-metabolism/abstract/S1550-4131(24)00079-2
- https://www.sciencedirect.com/science/article/pii/S2352396421003297
- https://www.ahajournals.org/doi/10.1161/HYPERTENSIONAHA.123.22250
- https://www.sciencedirect.com/science/article/abs/pii/S0923181117300269
- https://www.sciencedirect.com/science/article/pii/S1534580720302318
- https://karger.com/drm/article/239/4/533/836627/Cellular-Senescence-Ageing-and-Androgenetic
References
- https://pubmed.ncbi.nlm.nih.gov/23974577/
- https://pmc.ncbi.nlm.nih.gov/articles/PMC4908443/
- https://pmc.ncbi.nlm.nih.gov/articles/PMC5676194/
- https://www.ncbi.nlm.nih.gov/books/NBK430848/
- https://pmc.ncbi.nlm.nih.gov/articles/PMC11068553/
- https://pmc.ncbi.nlm.nih.gov/articles/PMC7310274/
- https://pubmed.ncbi.nlm.nih.gov/23385670/
- https://pubmed.ncbi.nlm.nih.gov/24248185/
- https://www.cell.com/cell-metabolism/abstract/S1550-4131(24)00079-2
- https://www.sciencedirect.com/science/article/pii/S2352396421003297
- https://www.ahajournals.org/doi/10.1161/HYPERTENSIONAHA.123.22250
- https://www.sciencedirect.com/science/article/abs/pii/S0923181117300269
- https://www.sciencedirect.com/science/article/pii/S1534580720302318
- https://karger.com/drm/article/239/4/533/836627/Cellular-Senescence-Ageing-and-Androgenetic
Reviewed by Alessandra Zonari, PhD, Chief Scientific Officer (CSO) and Co-Founder of OneSkin
Alessandra earned her Master’s degree in stem cell biology, and her PhD in skin regeneration and tissue engineering at the Federal University of Minas Gerais in Brazil in collaboration with the 3B’s Research Group in Portugal. Alessandra did a second post-doctoral at the University of Coimbra in Portugal. She is a co-inventor of three patents and has published 20 peer-reviewed papers in scientific journals.
Reviewed by Alessandra Zonari, PhD, Chief Scientific Officer (CSO) and Co-Founder of OneSkin
Alessandra earned her Master’s degree in stem cell biology, and her PhD in skin regeneration and tissue engineering at the Federal University of Minas Gerais in Brazil in collaboration with the 3B’s Research Group in Portugal. Alessandra did a second post-doctoral at the University of Coimbra in Portugal. She is a co-inventor of three patents and has published 20 peer-reviewed papers in scientific journals.
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Exploring the Difference Between Hydrating and Moi...
3 Natural Ways to Keep Your Skin Hydrated and Glow...
How To Create A Hydrating Skin Care Routine For Dr...
Senotherapeutics, Senolytics, and Senomorphics -- ...
Cleansing 101: How to Wash Your Face the Right Way...
Scientific Breakthrough: OneSkin’s scientists show...
Dry Skin 101: How To Identify This Skin Type
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Developing a Skin Care Routine for Normal Skin: 4 ...
What is Normal Skin & Do I Have This Skin Type?
A Step-by-Step Guide to Creating the Perfect Skin ...
Understanding Oily Skin: Causes & Characteristics
7 Keys to Understanding Menopause and Skin Changes
Is This The Root Cause of Aging?
UV Damage Increases Cellular Senescence. Here's Ho...
Our Research Has Been Published in npj Aging
How To Reduce Hyperpigmentation by Targeting Cellu...
4 Steps to Crafting the Perfect Skin Care Routine ...
A Comprehensive Guide to Understanding and Managin...
An In-Depth Look At The Benefits Of Moringa Oil Fo...
How to Tighten the Skin Under Your Eyes
7 Causes For Crepey Skin Under Eyes
101 Guide To Restoring Dry Skin Around The Eyes
How is OS-01 EYE different from OS-01 FACE?
How to Remove Lingering Dead Skin Cells On the Fac...
Safe Beauty, Validated: Why OneSkin trusts SkinSAF...
Hallmarks of Aging
What is Sensitive Skin?
How OS-01 Works on Mature Skin
Is Fragrance Bad for Your Skin?
Target Cellular Senescence with the Highest Concen...
Can a Plant-Based Diet Really Increase Your Health...
Eye Skin Ages Faster: Here’s How OS-01 Can Help
Gut Health and Skin: How Are They Connected?
The Science Behind Why The Skin Around the Eyes Ag...
How OS-01 EYE Supports the Ultra-Thin Skin Around ...
How Do You Know When to Stop Using Retinol?
How To Heal Skin From Picking Your Face Too Much
Collagen And Elastin: What Role Do They Play In Sk...
Why We Want Skin To Be More, Not Less
More Than Skin Deep: How Physical Touch Predicts L...
What is Skin Inflammation? What Causes it?
How to Reverse (or Prevent) Aging Skin
Yes, You Need Sunscreen During Winter
Can You Use Hyaluronic Acid with Retinol?
Exploring the skin’s purpose in whole-body health
Stressed Skin: 4 Stress Effects on the Skin
What Does Hyaluronic Acid Do For Your Skin?
Good Genes vs. Good Habits: Which Impacts Your Lon...
What Are The Causes Of Dry, Flaky Skin On Your Fac...
What is the Best Skin Care Regimen for Aging Skin?
7 Benefits of Jojoba Oil
What happens to skin as a person ages?
What are the Benefits of Sulforaphane?
8 Benefits of Andiroba Oil For Skin
Invasive vs. Non-Invasive Skincare: The pros and c...
Allantoin For Skin: A Comprehensive Guide
This is the Data to Look For When Choosing Skin Ca...
Why cellular senescence is more than just one of t...
A Simplified Guide To The Different Skin Texture T...
How OS-01 Works With Different Skin Types
How To Smooth Skin Texture On The Face: 4 Expert T...
Skin Microbiome: What It Is, Why it’s Important, A...
What Foods Cause Acne and Which Fight Against It?
The 101 Guide To Understanding Acne-Prone Skin
The Top 4 Prickly Pear Benefits for Skin
What Is Exfoliation & Is It A Necessary Measure Fo...
OneSkin Notable Mention: Dave Asprey's The Human U...
Retinol 101: Debunking Retinol as the Gold Standar...
Menopause & Skin: What you need to know
Take an Inside Look into Our Cutting-Edge R&D Plat...
101 Guide On The Benefits Of Cleansing Your Face
Cleanser Vs. Face Wash: Which Should I Reach For?
Five Common Skin Problems Experienced in Perimenop...
Get to Know OneSkin’s Topical Supplements:
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The Top 3 Benefits of Peptides for Skin
How to Use Peptides In Your Skincare Regimen to Op...
Peptides vs Retinol: Is One Better For Increasing ...
What Is Causing the Uneven Skin Tone On Your Body?
How To Boost Skin Moisture For Your Entire Body Fr...
Three Ways To Extend Your Skinspan Starting With Y...
What Are Free Radicals and How Do They Impact Skin...
The Top 6 Best Antioxidants For Skin Health and Lo...
How OneSkin Developed the OS-01 Peptide
How chronic stress makes you age faster
What is Epidermal Thickness & Why Does it Matter?
Cell Turnover & Why It Slows As We Age
OneSkin Notable Mention: OneSkin Featured In Tony ...
101 Guide To Skin Tightening + 7 Noninvasive Metho...
Tight Feeling Skin: 3 Reasons Why
3 Skin Tightening Ingredients To Help Boost Skin H...
How to Reduce Skin Inflammation & Redness
Niacinamide Vs Retinol: How Do They Compare?
How to Incorporate OneSkin’s OS-01 Topical Supplem...
Why Is Nutrient Absorption Key To Better Skin Heal...
OneSkin Notable Mention: LifeSpan Episode 6 | The ...
The Top 10 Best Foods for Skin Repair
Are There Dangers of Retinol Cream Use?
What is Skin Elasticity & How Can You Improve It?
A Complete Guide to the 4 Stages of Wound Healing
4 Foods To Avoid During Wound Healing & Why
Celebrating Black History Month: A Discussion With...
What is the Function of Skin as a Protective Barri...
Representation of the Black Community in Skin Care...
Our Story: Learn more about the history of OneSkin...
What Is Senescence & Its Relationship To Aging?
Special Announcement: OneSkin to Donate 1% of Sale...
Biological Age: What is Biological Age, Why Does i...
Peptides: The First Piece Of The Skin Longevity Pu...
Health Is The New Wealth
The Correlation Between Sleep and Skin: Why It Mat...
Calorie Restriction And Intermittent Fasting: Eati...
Healthspan: A New Way To Think About Aging